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A link between quantitative reductions in AGD and fetal androgen deprivation has been suggested in male reproductive disorders at birth (Dean and Sharpe 2013).
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The role of reactive oxygen species (ROS) in male infertility has been suggested in studies that found higher seminal ROS levels in infertile men compared with fertile controls (Agarwal et al. 1994; Pasqualotto et al. 2000).
Indeed, involvement of dosage compensation is suggested in the Spiroplasma-induced male killing in Drosophila [ 7].
A small increase in male births was suggested in that study, however, when both mothers and fathers were exposed to the highest category of PCBs--suggesting that paternal PCB exposure might be associated with the secondary sex ratio [ 52].
The idea that males have clitorises was suggested in 1987 by researcher Josephine Lowndes Sevely, who theorized that the male corpora cavernosa (a pair of sponge-like regions of erectile tissue which contain most of the blood in the penis during penile erection) are the true counterpart of the clitoris.
Further, it has been suggested that, in general, males adopt more problem-focused coping strategies and that females adopt more emotion-focused approaches [ 30].
Moreover, it has been suggested that in these species, males with territory defence mating tactic might preferentially increase their investment in post-copulatory traits to increase their fertilization efficiency whereas males with female defence mating tactic might increase their investment in pre-copulatory sexually selected traits to prevent other males from copulating with females.
It has been suggested that in the case of Jamaica, males are less likely than females to meet the place of residence criterion of sleeping at least three nights per week at the address [ 35].
Although the anti-inflammatory GC actions have been suggested to be more effective in males than in females [22], we did not find any sex-specific influence on hGRα mRNA and hGR-protein expressions, or on serum cortisol, HSP, and ILs concentrations.
Two factors have been suggested, the hemizygosity of X in males and dosage compensation [ 34, 50].
It has been suggested that increased enamel defects in males have been caused by increased nutritional requirements due to more rapid growth thus making males more susceptible than females to the formation of enamel defects.
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