The molecular mechanisms of regulating stem-cell proliferation have been studied in neural-, intestinal-, hematopoietic- and epithelial-stem cells of vertebrates and invertebrates (Tulina and Matunis, 2001; Barker et al., 2007; Ohlstein and Spradling 2007; Takashima et al., 2008; Farkas and Hutter, 2008; Reichert, 2011; Homem and Knoblich 2012).
Hematopoietic tissue damage was studied in 43 FA exposed workers, which were compared with 51 controls.
PYHIN genes have been predominantly studied in hematopoietic cells, and here we have compared expression of the twelve genes in splenic T and B cells, as well as in bone marrow-derived macrophages (BMM) and thioglycollate-elicited peritoneal macrophages.
Although Mcl-1 was initially studied in hematopoietic tumors, it is clear that many solid tumors are also dependent on this survival factor.
Several methods have been studied to obtain mature RBCs from CD34+ hematopoietic stem cells via in vitro culture.
Homeostasis in the hematopoietic system is studied by Bromodeoxyuridine (BrdU -labeling experiments to assess the degree of proliferation of newly generated cells.
However, cell lineage can be studied by over-expression or inactivation of some key transcriptional factors in hematopoietic lineages [ 58].
Chondroprogenitor and hematopoietic markers were also studied in different grades of OA.
Cross-talk between JAK/STAT5 and PI3-K pathways has been described in hematopoietic and non-hematopoietic cells.
ALDH1 expression has been found in hematopoietic and epithelial cancers.
