It has been shown that post-translational events substantially alter RXRα location by addressing the receptor to mitochondria, where it binds to mitochondrial DNA by forming heterodimerical complexes with p43, a truncated version of the triiodothyronine nuclear receptor [44].
It has been shown that post-vaccine antibodies wane faster in patients under immunosuppressive therapies than in healthy individuals [ 20, 21].
In addition to transcriptional regulation, it has been shown that post-transcriptional regulation also has a crucial role in modulating the expression of the CD154 gene.
It has been shown that post-CRT MRI can accurately predict tumours that are confined to the bowel wall (ypT0 2) with PPVs of 86 91% and NPVs of 70 75% [17].
We hypothesize that transposable elements are involved in LGD events in S. salar; it has been shown that post-WGD transposable element activity is highly genome specific in Brassica (Sarilar et al. 2013).
Recently it has been shown that post-discharge mortality in patients with heart failure increased in the last two decades while 30-day and in-hospital mortality rates decreased over time [ 3].
However, it has been shown that post-processing a larger number of BLAST hits can reveal useful taxonomic assignments, for example by using consensus information as implemented in the lowest common ancestor algorithm of MEGAN [ 60, 61] or the Darkhorse [ 62] and Kirsten [ 19] algorithms.
Previous studies have been showed that post-pretreatments and addition of some supplements increase the hydrophilicity of lignin [ 8- 10].
By analyzing several variants of a typical tubular truss bridge, it is shown that post-weld treatment can result in a significant fatigue performance improvement for this bridge type.
It is shown that post-oxidation by PIII leads to a more favourable corrosion protection with lower corrosion current and more positive corrosion potential compared to thermal oxidation or plasma oxidation.
It has been shown that in post-menopausal women with hormone-dependent breast cancer tumor proliferation is driven by increased levels of E2 [7]–[8].
