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It has recently been shown that modulation of STING can lead to an aggressive antitumor response.
It has previously been shown that modulation of γδT cells activates αβT cells resulting in marked T cell proliferation.
It has been shown that modulation of BER alters cellular sensitivity to UVA but not to UVB (Kim et al, 2002).
It has been shown that modulation of HMG-CoA synthase 1 activity influences cholesterol biosynthesis [37,38] and the enzyme is negatively regulated, via a feedback mechanism, in response to cholesterol feeding [39].
It has been shown that modulation of fibroblasts into myofibroblastic phenotype, with addition of particular contractile aspects, indispensable for connective tissue remodeling during the wound healing process in asthmatics airways [ 57].
It has been shown that modulation of cellular enzymes of gemcitabine transport and metabolism influences drug activity in vitro (Mackey et al, 1998, 1999; Goan et al, 1999; Ritzel et al, 2001).
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It is shown that modulation diversity improves the performance of digital communication systems, especially under severe fading.
It is shown that modulation of the string cross-section leads to a change of the eigenfrequencies as compared with their non-modulated values, and to the emergence of a spectrum of additional high eigenfrequencies, which correspond to large wave lengths.
Interestingly, recently was shown that modulation of the hippocampus-dependent memory by attention is dopamine-mediated [ 37].
Recently, it has been shown that this modulation can be exploited to select a very effective combination treatment of trabectedin followed by irinotecan (Grohar et al, 2013).
In agreement with this it has been shown that the modulation of auxin sensitivity under phosphate deficiency, responsible for the alteration in lateral root patterning, is dependent on TIR1 [ 19].
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