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It has been shown that increased knowledge about organ donation is associated with an increased likelihood of holding an organ donor card and feeling more comfortable in approaching relatives of potential organ donors [ 9].

Furthermore, it has been shown that increased VO2 peak is associated with decreased cardiovascular and all-cause mortality [38].

It has been shown that increased prostate size is a risk factor for lower urinary tract symptom (LUTS) progression in men who currently have LUTS presumed due to benign prostatic hyperplasia (BPH).

It has been shown that increased levels of aldosterone contribute to development of insulin resistance and thus act as a risk factor for the development of type-II diabetes mellitus.

In addition, it has been shown that increased levels of neutralizing endogenous serum GM-CSF autoantibodies (Ab) in CD patients as well as decreased expression of the GM-CSF receptor α-subunit (CD116) in circulating monocytes and granulocytes of CD patients affect mucosal integrity, epithelial barrier function, bacterial killing, and neutrophil antimicrobial functions (reviewed in [7]).

It has been shown that increased activity for recent negative trials occurs specifically after the onset of the probe item [15].

It has been shown that increased SBM formation in ladybird mis-expressing embryos occurs at the expense of the LT musculature, which is replaced by unfused mono-nucleated myoblasts [7].

Indeed it has been shown that increased susceptibility of mice to superinfection with Listeria monocytogenes after LCMV infection is caused by enhanced apoptosis of PMN leading to leukopenia and drastically reduced PMN infiltration at bacterial infection sites [22].

Similarly, it has also been shown that increased serum osteoprotegerin levels correlates with AACS [ 23].

It has been shown that increased surface expression of CRT is correlated with apoptosis rate.

Additionally, it has been shown that increased IKK activity promotes insulin resistance in obese mice when the kinase is overexpressed.

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