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In fact, it has been shown that developmental exposure of rats to PCBs results in severe hearing loss [31].
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Although, it has been shown that light regulates developmental processes at least in part through cross-talk with the phytohormone auxin [20], [21], the mechanism by which light and auxin interact to regulate root development is largely unknown.
However, it has been shown that during adolescence, developmental trajectories of white matter volume, as well as the MTR, differ between the sexes.
In recent "omics -based studies perfomics -basedze and other studies [ 44, 45, 18], it has been shown that the performedelonmaizel stand must be taken intotherount, aspecies are large differences both at the physiological and molecular levels during the transition from N assimilation to N remobilisation [ 18, 38].
However, it has been shown that microsatellite sequences in developmental genes are a source of variation in natural populations, affecting visible traits by expanding or contracting at very high rates [ 46].
It has been shown that, during a critical developmental period, the morphology of the dendritic tree deeply relies on the synaptic activity received by the dendrites (Spitzer, 2006; Cline and Haas, 2008).
It has been shown that the expression of several developmental genes is activated in epicardial cells during the early phase of heart regeneration, prior to myocardial regeneration and neo-vascularization (Lepilina et al., 2006).
It has previously been shown that in the mouse the developmental switching of globin gene expression correlates with the reconfiguration of an active chromatin hub (ACH), a complex of promoters of transcribed genes with distant regulatory elements.
It has been shown that autophagy is important during critical developmental stages in which nutrients are restricted.
Although senescence was originally identified as a stable cell cycle arrest, it has now been shown that senescence is also a programmed developmental mechanism that contributes toward mammalian embryonic development (Muñoz-Espín et al., 2013; Storer et al., 2013).
It has been shown that RACK1 plays regulatory roles in diverse developmental and physiological responses, including cell cycle control, cell movement and growth, immune response, and neural responses in mammals (reviewed in [ 3, 4]).
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