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It has been shown that binding of the amylosic chains to the starch binding domain alters the starch structure facilitating degradation by the catalytic domain.
It has been shown that binding affinity does not increase linear with the molecular size [2].
It has been shown that binding of the PcG protein Bmi1 to chromatin correlates with its phosphorylation status [58], [59].
It has been shown that binding of the anti-Alexa488 antibody to Alexa488 results in a fluorescence quench of Alexa488 [42].
It has been shown that binding of soluble P-selectin, expressed by ECs, to P-selectin glycoprotein ligand-1 (PSGL-1), expressed by CD34+ cells, activates cellular adhesion molecules on CD34+ cells, particularly αvβ5 integrins [18].
It has been shown that binding of one of these substrates (ferredoxin or NADP+) weakens the binding of the other.
Similar(41)
It was shown that binding of zinc insulin to PMAP is accompanied by conversion of the protein to its zinc-free form as a result of the Zn+2 release from the insulin native structure.
Recently, it was shown that binding of Asx to a well-characterized 25 kb sequence in the Ubx promoter region depends on trithorax (trx) [52], suggesting that Asx cooperates with trxG genes rather than PcG genes for Ubx regulation.
Using the testis-specific cerebroside sulfotransferase (CST) promoter and the IGF2/H19 imprinting control region (ICR), it was shown that binding of BORIS isoforms to DNA targets in vitro is methylation-sensitive and depends on the number and specific composition of ZF.
It was shown that binding of As III) into ArsA was greatly facilitated by the presence of magnesium ion and ATP.
Indeed, in rat hepatoma cells it could be shown that binding of SERBP1 protein to the PAI 1 mRNA leads to degradation and destabilisation of PAI 1 mRNA [ 3].
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com