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Furthermore, it has been reported that human dermal fibroblasts express receptors for β-glucans like macrophages.
It has been reported that human FOXP3 Tregs express surface LAP after activation [7], [8] by a GARP-mediated anchoring mechanism [8], [9].
In recent literature, it has been reported that human NB cells with MYCN amplification are selectively resistant to oxidative stress by transcriptionally up-regulating glutamate cysteine ligase [31].
For example, it has been reported that human Rspo1 synergizes with Wnt3a to stabilize β-catenin and that this synergy occurs in the extracellular environment [18], [32], [37].
It has also been reported that human CD4+ regulatory NKT cells can suppress the expansion of tumor antigen-specific CTLs via Th2 cytokines such as IL-4 and IL-10 [26].
Consistent with this it has recently been reported that human collagen bound to the GAS cell surface interacts with α2β1 integrins on endothelial cells inducing the uptake and transcytosis of GAS cells across endothelium [90].
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It has been reported that humans may ingest between 30 and 150 mg HMF daily via various food products; however, safe levels of HMF consumption are not well clarified.
Recently, it has been reported that human-specific protein-coding genes are often related to non-coding transcripts in macaque, pointing to a non-coding origin for many newly evolved proteins (Xie et al., 2012).
It was reported that human Hassall's corpuscles produce TSLP which activates thymic dendritic cells to upregulate expression of CD80 and CD86.
It was reported that human Ago2 bound to the cap through two conserved phenylalanine residues (F450 and F505) and thereby prevented the recruitment of eIF4E in miRNA repression (Kiriakidou et al., 2007).
It was reported that human EEF1A2 promoted cell growth and proliferation in human ovarian cancer.
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