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Although it has been reported that Cu nanofilament has been successfully formed in solid-electrolyte, Cu migration in solid-electrolyte has not been described in the literature to date.
It has been reported that Cu NPs have bactericidal effects comparable to Ag nanoparticles in single strains of E. coli and B. subtilis [ 10].
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It has been reported that high copper accumulation increases XIAP-Cu interaction, eventually leading to degradation of XIAP [ 22].
It has also been reported that other metal transporters can transport Cu into cells.
It has been reported that Cu II) in aqueous solution exists in different forms such as Cu2+, Cu(OH +, Cu(OH 2, Cu(OH) 3 − and Cu(OH 4 2− and the predominant copper species at pH < 6.0 is Cu2+ (Badruddoza et al. 2011; Xu et al. 2006).
We are interested in Ag/CuS nanocomposites because it has been reported that among the three metals (Ag, Au, Cu) with efficient plasmon resonances, Ag exhibits the highest efficiency of plasmon excitation (Evanoff and Chumanov 2005).
It has also been reported that (111 -oriented grains do not dominate in deposited Cu until pressures below 111 -oriented.
It has been reported that the combination of oxalic acid and chlorides in wall paintings results in Cu-hydroxychlorides Cu2Cl(OH)3 and Ca-oxalates [59]; and that no Cu-oxalates were observed in azurite paint layers [58].
In contrast, it has been reported that a preconditioning ischemia does not induce upregulation of SOD, whether MnSOD or Cu-ZnSOD [ 44].
It has been reported that mutations in Cu/Zn superoxide dismutase-1 (SOD1) can cause familial ALS.
However, it has also been reported that HDAC6 knockdown increases mutant Cu Zn superoxide dismutase (SOD1) aggregation in cultured cells and mutant G93A-SOD1 can modulate HDAC6 activity and increase tubulin acetylation.
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