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Recently, it has been reported that brain magnetic resonance imaging can reveal transient T2*-weighted abnormalities during the acute phase of migraine with aura.
More recently, it has been reported that brain magnetic resonance imaging (MRI) can reveal transient T2*-weighted abnormalities during the acute phase of MA [6 8].
It has been reported that brain CSCs are maintained within vascular niches [ 13].
It has been reported that brain capillary membranes that are isolated from hAPP mice exhibit a marked decrease in P-gp.
It has also been reported that brain samples from patients diagnosed with schizophrenia had lower levels of PERK and ATF4 than normal samples (Trinh et al., 2012).
In an experimental model for CM, it has been reported that brain EC purified from CM-susceptible and CM-resistant mice exhibit different sensitivities to TNF.
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It has also been reported that brain-tumour vessels lose their blood-brain barrier features after interaction with immune cells (Owens et al, 1994), and activated T cells cross the brain blood barrier of such vessels (Hafler and Weiner, 1987).
However, it has previously been reported that brains with AD have a lower percentage of euchromatin than control brains [ 105].
It was reported that brain cells are able to release pro-NGF into the extracellular space where the enzymatic conversion of pro-NGF to mature NGF form takes place (Lee et al. 2001; Bruno and Cuello 2006).
As early as 1972, it was reported that brain glycogen, residing almost exclusively in astrocytes [ 2], appears to be coupled with neuronal activation, with levels increasing during anaesthesia [ 3].
It has been reported that several brain diseases can be treated as transnosological manner implicating possible common molecular basis under those diseases.
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