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It has been reported that activated microglia kill neurons and blocking microglia Kv1.3 channels inhibits microglia neurotoxicity, indicating the involvement of Kv1.3 in microglia-mediated neurotoxicity.
It has also been reported that activated neutrophils undergo a peculiar cell death process different from apoptosis [45] which could escape detection by the kit used.
It has been reported that activated Pyk2 phosphorylates Hic-5 at tyrosine residue 60 resulting in up-regulation of LPA-mediated cell migration and the induction of EMT through a RhoA/ROCK-dependent pathway [17], [23], [24].
It has been reported that activated T cells strongly express RANKL in inflammatory regions [ 33, 34].
It has been reported that activated microglia express GLP-1, at least in culture [ 29].
It has also been reported that activated TLR4 signalling induces the expression of apoptotic molecules of the Fas death pathway [ 26].
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Recently, it has been reported that activating NK cell receptor ligands MICA (NKG2D ligand) and CD155 (DNAM-1 ligand) are differentially expressed during the progression to cervical cancer [ 15].
It is reported that Activate has closed down, again, less than two months after its official (re)raunch.
Consistent with the latter possibility, it has previously been reported that DC activated by yeast particles induce T cells with regulatory activity [ 29].
It has also been reported that phosphoramidites activated with 1H-tetrazole react, to a small extent, with the O6 position of guanosine.
Conversely, it had previously been reported that dnRAR activated msgn1 expression owing to dusp6 repression (Moreno and Kintner, 2004).
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been documented that activated
been demonstrated that activated
been described that activated
been determined that activated
been hypothesized that activated
been proposed that activated
been established that activated
been suggested that activated
been known that activated
been found that activated
been reported that activation
been shown that activated
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Justyna Jupowicz-Kozak
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