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On in vivo data, a standard error of 8.4% has been reported in bladder activity quantification [6].
Similar observations have been reported in bladder, breast and skin cancer [18], [29], [41] [44].
A deficiency in the decatenation checkpoint has been reported in bladder and lung cancer cells.
A similar approach has recently been reported in bladder carcinoma [ 25].
To our knowledge, this signature has not previously been reported in bladder cancers [ 16, 17, 20].
Low rates of amplification (15%) at the 11q13 region have been reported in bladder cancer (reviewed by Schuuring, 1995).
Similar(46)
Loss of expression of FABP4 was reported in bladder cancer while FABP1 and FABP2 are over-expressed in prostate and breast cancers [ 7, 31- 33].
Growth factors have been reported in the bladder development and the remodeling of the bladder wall after outlet obstruction [ 35].
Increased nerve growth factor (NGF) levels have been reported in the bladder tissue and urine of patients with overactive bladder and detrusor overactivity (DO).
In the case of PEITC, a sensitizing effect on Fas-mediated apoptosis has been reported in T24 bladder cancer cells and in Jurkat cells overexpressing Bcl-2.
Notably, there is evidence that Sprouty2 prevents Kras-induced lung cancer (Sutterluty et al., 2007), although such an effect has not yet been reported in human bladder cancer.
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