In previous studies, a pericholangial lymphocytic infiltration similar to that found in stage I of primary biliary cirrhosis, has been reported in 1°SS with abnormal liver function tests or positivity for AMA [ 4, 7].
The importance of central gray matter damage, thalamic atrophy, and cerebral white matter (WM) injury has also been reported in the context of abnormal neurodevelopment [ 6, 7].
RNAs encoding variant and mutant ERs have been reported in breast cancer [ 72]; abnormal receptors may bind oestrogens but not transmit a signal.
On the other hand, targets that have been reported in cancer studies, probably reflecting abnormal hERα activity at binding sites, exhibit many EREs but no SP1 motifs at promoters and few FOX sites along the transcripts (Table 2).
Abnormal phenotypes have been reported in BACE1-deficient mice, including a schizophrenia-like phenotype (Savonenko et al, 2008), abnormal muscle spindle formation (Cheret et al, 2013), and retinal pathology (Cai et al, 2012).
To our knowledge G morbillorum has not been reported in previous descriptions of normal or abnormal vaginal flora.
Abnormalities in secreted levels of vascular growth factors and soluble vascular receptors have been reported in patients with ASD [80], as have abnormal levels of the endothelial cell protein PECAM-1 (platelet endothelial cell adhesion molecule-1) [81].
Deficient vascular development and abnormal angiogenesis have been reported in women with HMB.
Abnormal EOGs have been reported in approximately a third of patients, and seem more frequent in patients with advanced disease.
It is however notable that a QTL for abnormal odor has been reported in the same chromosomal region (between markers SW1041-SW951) by Lee et al. [ 15].
