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Moreover, small molecule-induced cellularization has only been reported for mouse myotubes.
As has been reported for mouse DIA1 [64], we find both human DIA1 and DIA1R are ubiquitously expressed, suggesting each gene has a generic cellular role.
Our results are also consistent with the correlations between the juxtamembrane localization of actin fibers and decreased motility or decreased metastatic potential that have been reported for mouse embryonic fibroblasts [42] and for primary oral squamous cell carcinoma tumors [43], respectively.
Differences in responsiveness to PGE2 has, for example, also been reported for mouse chondroprogenitors and chondrocytes [ 39].
Furthermore, and also consistent with what has been reported for mouse adipocytes [ 16], a high percentage (49.5%) of the PPARγ binding sites overlap with C/EBPα binding sites.
Such behavior theoretically could underlie the observation of tumor-initiating cells in certain types of cancer (Reya et al, 2001) and has recently been reported for mouse intestinal stem cells (Vermeulen et al, 2013; Snippert et al, 2014).
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As a result, no recent estimates of the rate constants obtained using a state-of-the-art high-resolution scanner are available for human studies (estimates have only been reported for mice; see [8]).
Moreover, results similar to those of Cinque et al. [15] have been reported for mice infected with H. felis [32] [34].
A similar deficiency in liver regeneration has been reported for mice lacking the nuclear receptor FXR which are also prone for spontaneous hepatocellular carcinoma [79], [80].
Similar results have been reported for mice exposed to diethylstilbestrol during development.
Only one judgement bias study has been reported for mice where odour cues were used to predict rewarding or aversive food outcomes (Boleij et al., 2012; Table 2014).
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