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Although it has recently been recognised that inflammation is important in the development of work-related musculoskeletal disorders (MSDs), the exact pathophysiological pathways are unknown.
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In humans, it has long been recognised that it is poor regulation of inflammation that contributes to poorer reproductive outcomes [ 65, 66].
It has been recognised that alterations in lipid metabolism can lead to cancer, cardiovascular disease, diabetes, neurodegenerative disorders, immune function, pain, mental disorders and inflammation.
Indeed, it is recognised that tissue hypoxia is a potent stimulator of local and systemic inflammation (Wouters, 2005; Zinkernagel et al, 2007).
The role of inflammation in the generation of arrhythmias in HF and following ischaemic injury is not clear, but it is recognised that malignant arrhythmias are more likely in patients with acute and chronic myocarditis [ 144, 145].
It is recognised that cubical particles provide more realistic results.
It is recognised that cooling systems need to be flexible.
It is recognised that coal is a porous solid.
We are recognising that joblessness is a crisis.
It has been increasingly recognised that inflammation in general, and macrophages in particular, may play significant roles in prostate carcinogenesis and cancer progression.
Furthermore, crystalline cholesterol found in atherosclerotic plaques, and serum amyloid A, an acute phase response protein in mice, have been recognised as DAMPs that exacerbate inflammation (Duewell et al., 2010; Niemi et al., 2011).
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