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For the development of our framework, we formally extend the concept ofcounterexample, which has been proposed in model checking previously, and provide examples which demonstrate the need for such an extension.
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As has been proposed in models of facet fatigue formation, an example is presented of the accumulation, within a soft grain, of GNDs in a diffuse pile-up against a grain boundary with a hard grain.
Other models have been proposed in modeling seasonal variation, including generalized additive models, introduced by Hastie and Tibshirani, 1994, [ 12, 17, 49], and classic time series analysis [ 15, 16, 27, 50, 51].
It has been proposed in models of the lifecycle of DNA-mediated TEs [ 5- 7] that most TEs will eventually become inactivated in a given species, which underscores the importance of horizontal transfer for TE survival, a mechanism that allows TEs to invade a naïve genome.
The most recent bond-slip relationship is proposed in Model Code 2010 20122) and allows distinguishing between pull-out and splitting failure, and in this last case, only, allows accounting for the stirrups presence.
Moreover, its interaction prior is similar to the ones that have been proposed in earlier models that did not model causal inference but only the interaction between cues [13], [14], [17], [18].
A similar role for mitochondria-mediated oxidative stress has been proposed in a model for the aging process (Lenaz 1998).
Several forms of them have been proposed in standard models, and there has been a significant interest in developing numerical schemes for their solutions.
Similar to M1/M2 polarization a paradigm of N1 (antitumor) and N2 (protumor) responses has been proposed in murine models of lung cancer [104].
Such a pattern of peaked inflammation followed by low levels of sustained inflammation has recently been proposed in mammalian models of infection [15].
Various combinations have been proposed in various models in the literature.
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