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It has been postulated that prostate cancer is more heterogeneous and innately drug resistant, that it afflicts older and less robust patients, or that the impact of neuroendocrine cells confounds the assessment of outcome.
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It has been postulated that castration-resistant prostate cancer (CRPC) commonly remains hormone dependent.
Although the mechanisms for the resistance are unclear, it has been postulated that the development of tumors, such as refractory prostate cancer, is related to the accumulation of genetic or epigenetic alterations.
It has been postulated that vitamins D and soy may have a protective effect against prostate cancer, whereas there is a controversy around the protective effect of vitamin E and selenium.
It has recently been postulated that α5 may be crucial for cell detachment and subsequent metastasis of prostate cancer (Neal et al, 2011), which is in line with our results.
It has been postulated that 2-hydroxylated estradiol is anticarcinogenic.
It has been postulated that caspase-2 can directly injure mitochondria.
It has been postulated that bisphosphonates may affect healing if given soon after a fracture.
It has been postulated that androgens, through their interaction with androgen receptors, may play an important role in the development of specific tumors, such as ovarian and prostate cancer [ 16, 17].
It is postulated that this method may better accommodate the age-related increases in PSA level that occur independently of prostate cancer and thus distinguish deviations from the usual pattern in men with cancer.
It is postulated that PACAP is involved in nociception.
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com