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Decreased blood flow by Doppler might indicate irreversible disease, and it has been postulated that inflammation can initially manifest with increased blood flow.
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Although it has been postulated that systemic inflammation, malnutrition, and impaired renal function play a significant role in the underlying pathological processes [ 13], the mechanism of the association between increased RDW and mortality remains unclear.
It has been postulated that the progressive inflammation in the aorta of TA patients can be due to a chronic infectious process [ 7].
Rheumatoid arthritis (RA) is associated with an increased risk for cardiovascular disease (CVD), and it has been postulated that RA disease-related inflammation contributes to endothelial dysfunction.
12 13 It has been postulated that the reduction in inflammation afforded by both MTX and anti-TNF therapies can lead to the reduction in cardiovascular events as shown in other studies.
Recently, it has been postulated that intermittent hypoxia can induce inflammation and that the development of inflammation in response to hypoxia may be clinically relevant.
Inflammation is an early physiological response to fracture and it has been postulated that signaling molecules produced during inflammation initiate the tissue regeneration pathway (Mountziaris and Mikos 2008, Cottrell et al. 2009).
It has been postulated that it is the severity of inflammation and duration of the rheumatoid arthritis that contributes to the increased risk of lymphoma (Askling et al. 2009).
It has been postulated that the common link among these conditions is inflammation.
In RA it has been postulated that bone destruction occurs as a direct consequence of inflammation.
Although it has been postulated that wastewater containing dispersed MWCNTs can cause abnormal apoptosis and inflammation in aquatic species, only their toxicity was studied [ 11, 38– 42].
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