Similar anterior negativities with rightward lateralization (RAN) have been observed in response to "syntactic" violations during music processing (Koelsch, Gunter, Wittfoth, & Sammler, 2005; Patel et al., 1998).
Furthermore, increased EPCs levels have been observed in response to acute bouts of exercise in healthy populations [12 14] and patients with CHF [5].
Pronounced changes in the miRNA profile have also been observed in response to hyperoxia in neonatal lungs during experimental development of BPD [39, 40].
Induction of ER stress in the lung has been observed in response to cigarette smoke exposure as well as in pulmonary fibrosis [62].
Event related desynchronization (ERD) of mu waves, or mu suppression, over sensorimotor cortex has been observed in response to self-generated movement, viewing movement, or imaging movement.
Evidence of genotoxicity has been previously researched within a number of studies; micronuclei development is associated with nano-TiO2 exposure, which is indicative of chromosomal damage; DNA damage has also been observed in response to nano-TiO2 exposure.
K63-linked ubiquitination of histones has been observed in response to DNA damage [93] [95].
These morphological changes are similar to what has been observed in response to heparan sulfate.
However, it has since been observed in response to a range of stimuli including sinusoidal [28] and complex tones [29].
Also alterations in islet size, number and composition have been observed in response to nutritional changes [10].
Specifically, pharmacologic inhibition of CDKs in neurons affects activity of AP-1 and direct phosphorylation of c-Jun by CDK3 has been observed in response to erythropoietin receptor signaling in epithelial cells [23], [24].
