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Dietary factors have long been implicated in acne pathogenesis.
In addition to the pathogenic factors from the classical model, altered sebum lipid quality, regulation of steroidogenesis in the skin, interaction with neuropeptidases, androgen activity, nutrition, and the presence of any pro- and anti-inflammatory agents have also been implicated in acne development.
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Glycemic index (GI) and glycemic load (GL) may be implicated in acne pathogenesis.
The anti-milk lobby also claims that consumption of dairy products can aggravate rheumatoid arthritis and has been implicated in colic, acne, heart disease, asthma, lymphoma, ovarian cancer and multiple sclerosis.
In addition non-sense and truncating mutations in the genes of the γ-secretase complex, including Nicastrin have recently been implicated in Familial Acne Inversa and haploinsufficency was inferred to be causal.
Propionibacterium acnes has long been implicated in the pathogenesis of inflammatory acne [ 1, 2].
PGE2 has been implicated in the pathogenesis of acne and has also been suggested to be a viable target for the treatment of this disease [ 28, 33, 34].
Chocolate has been implicated in causing a litany of problems including acne and obesity.
McDowell has been implicated in that dispute.
APHs have been implicated in antibiotic resistance.
APOE has been implicated in Aβ clearance.
More suggestions(16)
been implicated in metastasis
been implicated in autism
been implicated in regulation
been implicated in heterochromatin
been implicated in depression
been implicated in immunity
been implicated in glioma
been implicated in chemoresistance
been implicated in lifespan
been implicated in tumorigenesis
been implicated in mechanotransduction
been implicated in malignancy
been implicated in tumour
been implicated in lung
been implicated in apoptosis
been implicated in growth
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