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Lung damage and changes in physiological state directly caused by influenza virus have also been implicated as promoting coinfection, typically using highly pathogenic viral strains.
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By extension, the psychologists who are members of the association are implicated as well.
Arachadonic acid and its metabolites such as 5-HETE have been implicated as growth promoting factors for various human cancers (Avis et al, 1996; Ghosh and Myers, 1998; Ding et al, 1999a).
IGF-1 has been implicated as a factor promoting hypertrophy in the adult animal.
In addition, physical exercise has been implicated as a factor promoting development and progression of the ARVC/D phenotype.
5-hydrocicosatetraenoic acid (5-HETE), the major 5-lipoxygenase metabolite of arachidonic acid, has been implicated as a growth promoting factor for various human cancer cells including prostate, lung and pancreatic cancer cells (Avis et al, 1996; Ghosh and Myers, 1998; Ding et al, 1999a).
Predation has been implicated as a selective force promoting divergence among taxa in only a few animal lineages.
Several viruses, especially gammaherpesviruses, have been implicated as co-factors for initiating, promoting or exacerbating IPF [ 14, 25- 27].
These studies have led to the idea that miRNAs may have a promising therapeutic aspect, since miRNAs have been implicated as both the active agent promoting regeneration, and as the regulatory mediator helping to uncover downstream targets and transcription factors involved in regeneration.
The dynamic history of coral reefs associated with sea-level fluctuations from the mid Miocene to the end of the Pleistocene has been implicated as the major evolutionary force promoting divergence and subsequent coalescence in species with high dispersal potential [e.g. [ 51, 76, 82]].
Aside from exerting major anti-fibrotic effects, PGE2 has recently been implicated as playing a central role in promoting the 'apoptosis paradox' of IPF.
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