Various Mre11 mutations have been identified in several types of cancer.
Critically, minimal essential subsets of hydrolases have now been identified in several model organisms, enabling the reduction of genetic complexity.
Pathogenic gene fusions have been identified in several histologic types of salivary gland neoplasia, but not previously in acinic cell carcinoma (AcCC).
Among them, activation of EGFR has been identified in several independent studies.
The use of beeswax as a binder or a coating has been identified in several examples [37, 38].
MELK overexpression has been identified in several human cancers: prostate [26], breast [27], brain [28], colorectal [29] and gastric [8].
This same motif has been identified in several other genes and in some also serves as a binding site for transcription regulatory factors.
Octopamine has been identified in several organs of normal rats by means of a sensitive enzymatic assay.
Candidate tissue-specific stem cells have been identified in several tissues based on the SP phenotype.
Other mutations have also been identified in several other genes [4] [9].
The T58A mutation has been identified in several cases of Burkitt's lymphomas.
