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Gene expression changes in brains of individuals with schizophrenia (SZ) have been hypothesized to reflect possible pathways related to pathophysiology and/or medication.
This association has been hypothesized to reflect alterations in neural circuits supporting bottom-up threat processing and top-down executive control.
This activity precedes awareness of the ensuing movement by as much as two seconds and has been hypothesized to reflect preconscious planning and/or preparation of the movement.
Common activations in prefrontal cortex (PFC) during episodic and semantic long-term memory (LTM) tasks have been hypothesized to reflect functional overlap in terms of working memory (WM) and cognitive control.
High VMAT2 expression has also been found in insulin-producing beta cells in the endocrine islets of Langerhans, but not in the exocrine pancreas [3], and the regional receptor density in the pancreas has therefore been hypothesized to reflect actual beta cell mass [BCM].
Note that likewise the facing bias for orthographic plws has been hypothesized to reflect evolved perceptual biases [30], [33].
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This disruption is hypothesized to reflect abnormalities in sensorimotor gating.
A unique clustering of layered streamlined forms in Athabasca Valles is hypothesized to reflect a significant hydraulic event.
These complex rhythms are hypothesized to reflect interactions between spontaneous pacemakers, wave propagation, refractory period, and overdrive suppression.
Different strain magnitudes in different areas of the same animal are hypothesized to reflect optimization to different criteria.
GMC excess in ipsilateral parahippocampal, cerebellar, and pericallosal regions was common to both left and right HA groups relative to controls, and is hypothesized to reflect diminished gray white matter demarcation, underlying white matter atrophy, or structural displacement due to cerebrospinal fluid expansion.
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