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The underlying mechanism of MTX-induced hepatotoxicity has not been fully identified, however, several distinct mechanisms have been proposed including cellular antioxidant defense deregulation which cause more oxidative stress-induced damages to the liver cells [ 7].
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Nevertheless, this effect has not been fully identified yet.
However, the detailed mechanisms underlying these effects have not yet been fully identified.
However, the sets of genes required for adaptation to low temperatures have not yet been fully identified [ 10].
However, the precise mechanisms that produce an elevated tCho concentration ([tCho]) have not yet been fully identified.
However, the causes of sustained ERK activation and p38MAPK inactivation in RMS, and the downstream effectors of MAPKs that elicit specific responses, have not yet been fully identified.
However, not all disease-risk genes have been fully identified.
However, the factors that contribute to renal fibrosis have not been fully identified.
However, the mechanisms responsible for such an association have not been fully identified.
However, the intracellular molecular mechanism triggered by C5a in neutrophils has not been fully identified.
However, endogenous receptors for mycolic acids on susceptible cells exposed to mycobacterial infections have not been fully identified.
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CEO of Professional Science Editing for Scientists @ prosciediting.com