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Glucose toxicity has been explained by increased cellular glucose flux and mitochondrially generated oxidative stress.
Associations between herd size and disease were also found in other studies and have been explained by increased stocking density and less time for individual care of calves [ 8, 9].
This finding is in agreement with studies from high- and middle-income countries where rising CS rates have not been explained by increased risk indication [ 7, 9] and another study from northern Tanzania that revealed many CSs done on questionable indications [ 16].
Effects of Doxorubicin, an antracycline extensively used in breast cancer treatment, have been examined in a number of animal studies, and the myotoxic effect has in part been explained by increased oxidative stress by reactive oxygen species (ROS) causing muscular dysfunction [ 41, 42].
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The increase in prostaglandins may be explained by increased cyclooxygenase expression or increased cyclooxygenase catalytic activity.
This reduction can be explained by increased environmental stability and reveals the existence of structural variability within physiographic types.
To some, it has all the hallmarks of an epidemic; others say it can in large part be explained by increased public awareness.
Mutant coefficient of coincidence and crossover number phenotypes cannot be explained by increased DSBs or by prolongation of the crossover-designation stage.
The generally larger pores in the butyl-modified chitosan scaffolds might be explained by increased phase separation rates due to the introduced hydrophobicity of the chitosan polymer.
Part of this association could be explained by increased risk of fatal ventricular arrhythmias [15].
The mitigating effect of CO2 is explained by increased crop water productivity (CWP), reflecting higher crop yield and less evapotranspiration, the loss of water to the air.
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been paralleled by increased
been disrupted by increased
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com