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Murine models of lupus (conventional or engineered) have yielded interesting snapshots of the signaling status of lupus lymphocytes, and many of these alterations in cell signaling observed in murine models of lupus have also been documented in patient samples.
Over-expression of EGFR relative to normal kidney tissue has been documented in patient RCC samples, and is thought to be associated with the transformation of normal renal tissue to malignancy [ 28].
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A hypercoagulable stable has been documented in patients with β-thalassemia.
Non-specific disturbances on the dopaminergic system have been documented in patients with BMS [8, 9].
These structural changes have also been documented in patients with migraine [10 13].
Abnormal somatosensory evoked potentials (SEPs) have been documented in patients with adolescent idiopathic scoliosis (AIS) with different cure severity.
Alterations in tissue zinc levels have been documented in patients with gastrointestinal tract malignancies and more frequently, in those with colonic cancer.
This apparent polymorphism of bloodstream strains has previously been documented in patients for opa and pilin expression [61].
Cognitive deficits have been documented in patients with bipolar disorder.
In fact, radiographic progression of joint damage has been documented in patients who were in clinical remission [ 4].
As of June 2002, 8 VISA infections had been documented in patients in the US[ 5].
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