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This commercially available antibody has been described to detect endogenous levels of either individually or dually phosphorylated human p44 and p42 MAP kinases phosphorylated at Thr202 and Tyr204 of p44 and Thr185 and Tyr187 of p42.
Regarding in vitro tests, an egg hatch assay (EHA) has been described to detect BZ resistance in Trichostrongylidae [ 14, 15].
Recently, the use of EdU has been described to detect DNA replication in vivo and in vitro in animals.
Strategies have been described to detect remote homology [ 45], i.e., fold conservation between proteins that share weak primary sequence similarity.
The primary objective of this systematic review is to identify which antibodies have previously been described to detect TDP-43 pathology.
The assumption of a pure-birth process (μ = 0) simplifies equation (3) as described by Kendall [ 29] and Nee et al. [ 9], and a likelihood-based approach has been described to detect shifts in diversification rates through time [ 12].
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Because MusPV may have been missed during routine pathogen screening of mice in colonies worldwide, a variety of detection methods are described to detect MusPV.
A Bayesian change point model is described to detect changes in temporal rates.
Recently, a FMDV microarray was described to detect simultaneously the seven FMDV serotypes.
The SMI-31 antibody is directed against the phophorylated KSP repeats in the C-tail of NFH and to a lesser extent NFM, while the SMI-33 antibody is described to detect non-phosphorylated NFH and to a lesser extent NFM [ 49, 53, 60].
The calf compression test (squeeze test) [ 2], the External Rotation test [ 1], and the direct eversion maneuver have all been described to clinically detect syndesmotic instability, with sensitivity ranging from 33-92% [ 3].
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