Overall enhancement in EEC polymerization rates and ultimate conversion were attributed to activated monomer mechanism associated with EPOH terminal hydroxyl groups.
It potentially could be attributed to activation in S1 and S2 and recorded together, since both are activated by noxious stimuli [13].
The tactile and cold allodynia induced by paclitaxel have been attributed to its ability to activate both TRPV4 and TRPA1.
Fibrinogen depletion has also been shown to reduce the severity of EAE, and this effect has been attributed to interference with its role in activating resident microglial cells [ 2].
Function of micF has been attributed to down-regulate OMPs [34] and is activated by marA, rob and sox [35].
In contrast, only a few experimental studies have demonstrated xenon's pro-inflammatory properties, which have been attributed to its leucocyte- and platelet-activating properties [ 6] and/or influence on Ca2+ homeostasis [ 5].
This buffer-evoked response has been attributed to the physical force of agonist application activating mechanosensitive ion channels [18,22].
For example, a "nitric oxide activated deoxygenase" function has been attributed to Ascaris Hb based on in vitro experiments using NADPH at pH 6.0 to achieve reduction [36].
This has been attributed to negative regulation by transforming growth factor beta-1, which is specifically activated in the extracellular matrix of ERα-positive cells [ 66].
The slowly activating anion conductance in guard cells has been attributed to AtSLAC1 and AtSLAH3 channels [ 24, 78], while guard cell QUAC is mediated by AtALMT12 [ 79].
