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This would fit into prolonged disease history in MAC because low BMI in NTM disease has been associated to abnormal control of leptin and adipokines, suggesting a prolonged disease history [ 30, 32].
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aHCF were found at the nuclear periphery and are associated to abnormal spatial positioning and expression of GMCL1.
In some cases, different behaviours during the progamic phase were associated to abnormal embryo development [ 13, 16].
Similarly, IVMS aneurysm has been associated to congenitally abnormal positioning of the aorta.
Partial AZFc deletions were shown not to be exclusively associated to abnormal sperm concentrations but significantly so to infertility [ 17, 19, 20, 25, 27, 28], thus selecting controls based solely in normal sperm counts may be a considerable confounding factor.
Maternal exposure to pollutants has been associated with abnormal placental vascular function, and consequently, fetal growth restriction and LBW [ 35– 35].
iPLA2γ-deficiency has been associated with abnormal mitochondrial function.
Cardiac hypertrophy, fibrosis, and ventricular dysfunction have been associated with abnormal levels of sST2 [38] 38].
Impaired working memory (WM) function in schizophrenia has been associated with abnormal activation of the dorsolateral prefrontal cortex (DLPFC).
Decreased blood hemoglobin (HbB) levels and anemia have been associated with abnormal brainstem auditory evoked responses (BAER).
Reln expression has recently been associated with abnormal bone remodelling of the otic capsule in the pathogenesis of otosclerosis [41].
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