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We include the overdominant model because we observe that heterozygous individuals sometimes exhibit extreme phenotypes.
Because we observe that strong negative feedback increases variability and so is unlikely to be a mechanism for noise control, we suggest instead that negative feedback is evolutionarily favoured because it allows the cell to minimize mRNA usage.
Our findings agree with these observations, because we observe that the Wg effect is best observed when hh is absent, suggesting that when the hh gene is present a boundary may be formed, thus preventing Wg from orienting the denticles to the same extent.
We observe that the part of the maximal achievable rate region which is unachievable by TS and SM is bigger when M is small because we observe that for the case of 4-PAM, we have one configuration for SM.
Because we observe that Se enhances their contact without changing the levels of these proteins, we propose that this lipid promotes the closed conformation of syntaxin1, which has the highest affinity for Munc18 [22].
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The difference in our technique is that image denoising is performed prior to upsampling because we observed that performing denoising after upsampling did not yield observable differences.
Furthermore, the beta distribution was justified because we observed that the depths follow a negative binomial distribution, and hence the observed allele frequencies are a ratio of such distributions, which is approximated well by a beta distribution.
Note that we use the Euclidean square (and not the disk) because we observed that our numerical simulations worked better in this case.
Because we observed that the gradiometry estimation outside of the station network was very poor, we limited the distribution of grid points to locations inside of Delaunay triangles having Hi-net station locations as triangle nodes.
We divided hyperchloremia into two categories because we observed that the mean chloride level in our population (107.3 ± 5.5 mmol/L) was at the high end of our normal range for chloride.
Because we observed that LRR1 expression is required for XA21-mediated immunity, we examined if overexpression of LRR1 is sufficient to enhance resistance to PXO99 in the rice variety Kitaake, which lacks Xa21.
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