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This was because the previous analysis showed that variation in the measured diclofenac concentration among sub-samples taken from the liver of the same ungulate had a negligible effect on the outcome of the analysis.
Because the previous analysis of HSF binding events on polytene chromosomes was not of sufficient resolution to determine which genes HSF associated with at heat shock temperatures on a global scale, we set out to determine what genes may be affected by HSF binding by identifying the genes closest to each HSF binding site.
Because the previous analysis revealed significant differences in patient satisfaction among the five health districts, we also examined the evolution in patient satisfaction for each district separately.
We restricted these analyses to the dHPC because the previous analysis found no significant increase of vHPC above baseline during the radial maze tasks.
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This analysis was even more conservative than the previous analysis, because each of the covariates (stimulus type, emotion condition, response accuracy, etc).
Step 1 of the procedure required a more elaborate treatment than that used in the previous analysis because the present study compares data from three surveys rather than reporting just one.
This analytical approach differs from the previous analysis because the crowd out phenomenon is stochastic and contingent on the concurrence of other disease outbreaks.
An estimated proportion of invariant sites was not used in the previous analysis because it had not been implemented in the used version of Bio++.
This is likely because interindividual variability was not considered in the previous analysis.
They were, as opposed to the previous analysis, not included in the fixed model because the HHI was measured per diagnosis.
The sequences of Ammotragus were not included in this analysis because of the inconsistence of its phylogenetic position that we found in the previous analysis.
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