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Because the maps presented here are gene-based, they can be easily associated with other gene-based genetic maps regardless of the polymorphic positon(s) within the gene.
This is because the maps presented predict the probability of disease presence in an area, rather than directly infer measures of incidence or burden, which can be influenced by a variety of other factors (e.g., in the Mediterranean coastline of Europe, VL has been associated with immunosuppression).
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Because the marker density of the maps presented here was higher than that used to assign and order the sequence scaffolds in the Btau_2.0 genome assembly, we were able to position additional scaffolds.
Nevertheless, the map presented here has markers positioned with very high confidence, and, because it contains no cosegregating markers, it is better suited to QTL detection.
There is reasonable consistency between the RH map presented here, the MARC 2004 linkage map and the ILTX 2005 map.
To our knowledge, the genetic map presented in this paper is the densest map to date for sesame, though it is still not saturated, because it has 15 LGs, not 13 LGs as was expected.
Because this manipulation introduced additional uncertainties, we decided to limit the updated map presented here to genes with mutant phenotypes mapped relative to each other.
The multimedia map presented in Sect.
Because the results are presented as maps that show the net changes in conditions, they are accessible by a wide audience.
Because EMAP ontologies were recently updated to the EMAPA ontology (34), we mapped the presented brain regions also to these identifiers (Supplementary Table S1).
The chromosome map of LG7 featuring RH and FISH maps along with the genetic map is presented in Figure 3.
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