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Because the expected gene has been retired, the target genes of these strains may be localized in intergenic regions or they could be any other genes, as the sequencing results indicated.
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Because we expected genes relevant to type 2 diabetes to have a high Δrank compared with background, we ordered each quantile by Δrank, then split each set into those above and below the median Δrank for that quantile.
This is not surprising, because we expect genes that function in basic cellular processes to be essential and are best captured using balancer systems.
Because the expected higher translation efficiencies of these genes is due to increased elongation rates, and if the initiation rate is the same, the increased elongation rate should decrease ribosome density.
Now that naturally occurring human genes can't be patented, expect gene testing companies to benefit broadly with lower-cost products across the board.
The two lists do not show any significant overlap for any of the ranks, because, as expected, the proportion of genes in the overlaps (y-axis) is roughly equal to the proportion of genes selected (x-axis).
Consequently, prediction of diversity in the ungenotyped regions between markers is more relevant than the expected diversity at the markers, because most genes of interest will be in the regions between two markers.
Because we expect the gene pairs to be products of duplication, we did not require them to be mutual best BLAST hits.
ABCD3 staining was observed predominantly in the peroxisomal membrane of noncancerous and prostate cancer tissues, which was expected because the ABCD3 gene product, PMP70, is a membrane bound peroxisomal protein.
Given the complex inheritance pattern of autism, it is expected that gene-gene interactions will exist.
This observation was expected because gene deletions on the X chromosome are more likely to be deleterious than amplifications.
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CEO of Professional Science Editing for Scientists @ prosciediting.com