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We used quantitative RT-PCR (qPCR) to show upregulation of FAS II genes in liver stages because our previous microarray analysis had indicated preferential expression in liver stages of P. yoelii (Tarun et al., 2008).
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Our previous microarray analysis revealed that P311 was highly upregulated in hypertrophic scar tissue [3].
Our previous microarray data [6] showed significant up-regulation of ECT2 in OSCC-derived cell lines.
Our previous microarray analysis of Maestro utilised longer spotted cDNA probes [9].
CSHL1 is one of the potentially pregnancy-specific placental expressed transcripts identified in our previous microarray study [6].
Our previous microarray study [6] showed that these transcripts had much higher expression in placental tissues than blood cells.
We have shown the clinical utility of P63 IHC for the identification of lung SCCs, further validating our previous microarray study.
Through a comparative analysis with our previous microarray data, we could also identify with high confidence genes specifically induced by one pathogen.
Our previous microarray analysis [2] identified nfrA, coding for nitro/flavin reductase [43], [44], as one of the genes activated by Spx.
This finding was consistent with our previous microarray study [11] which suggested that viral load was a determining factor in Sn expression.
Our previous microarray analysis indicated that miR-34c was downregulated in nasopharyngeal carcinoma (NPC).
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