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Because of this deficiency, host-based IDS solutions have been introduced [4, 5].
Because of this deficiency, the tests performed in the present study, as well as the 2D full-scale laboratory tests by Lew et al. (2011), have been used to calibrate the numerical models.
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Because of this transporter deficiency, this strain is unable to grow on D-xylose and is severely defective in its growth on D-glucose.
We acknowledge the importance of this deficiency because researchers in our network act under different circumstances and we know that this diversity has a real impact on the structure and eventually on the dynamics of the network, as well as on the results of our model.
Because of the deficiency in xylose transport, this strain is unable to grow on xylose.
This is mostly because of the deficiency of Pearson in the detection of the coresponse of pairs that contains lags in the time courses (see Supplementary Fig. S2 for evaluation of only lagged time courses).
The precise mechanisms underlying this phenomenon are poorly understood, partly because of the deficiency of research-related findings.
However, experimental methods to monitor this process in mammalian cells are limited because of the deficiency of autophagic markers.
However, the precise mechanisms underlying synergistic relationship between OFP and headache are poorly understood, partly because of the deficiency of research-related findings.
Additionally, 3 studies were further excluded because of the deficiency of data integrity.
Depletion of DHODH is reported to trigger a p53 response because of the deficiency in pyrimidine [ 13].
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