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This should hold true because, in both models, the physician will treat all high priority before low priority patients and delegates will only treat low priority patients.
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This occurs because B∞ in both models asymptotically goes to zero as 1/τ1.
Any of these variables did not change effect estimates >10%, and therefore in the results we only show the multivariable model 1 because the results in both models were almost identical.
The classical solution is much better, because in classical models, pervasive systematicity comes for free.
This is because, in these models, the load was applied axially (that is, as a uniform displacement).
However, one would then expect similar results in both ossification models, because the carriers used in both models were identical.
Correlations between EBV from SEXTLM and GDM dropped to around minus 0.90 because the assumptions made in both models were different.
Because the variation remains significant in both models, other predictors still remain to be found.
In particular, until about generation 80, all cells in both models divide because no cells have yet become senescent.
Because the ventricular structures are identical in both models, we can visualize differences in activation by plotting the difference in activation times at each point in the ventricles.
The neural network was therefore applied to model the relationship because, in principle, this model accounts for various implicit or explicit relationships [ 28, 29].
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com