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Our combined modelling approach provides a reasonably good fit with the observed data for the spread of faba bean rust.
The model was fitted to data for the spatio-temporal spread of faba bean rust (caused by Uromyces viciae-fabae) through a discontinuous field.
From the outset the Bean/Cowpea Collaborative Research Support Program (CRSP) has emphasized integrated disease management, and breeding for resistance to bean rust (Uromyces appendiculatus), common bacterial blight (Xanthomonas campestris (=X. axonopodis) pv.
Consequently, yield losses due to fungal infection, like Uromyces appendiculatus (bean rust), have strong consequences.
Regulation of selected bean genes in response to bean rust infection was confirmed by qRT-PCR.
Several resistant genes were identified that confer resistance to bean rust infection.
Similar(29)
As seen in the comparison to the Arabidopsis transcriptome, the most abundant category was comprised of 30.13% of the unigenes with unknown functions which was consistent with the previous study by Thibivilliers et al. [ 7], who found that 31.9% of common bean ESTs from bean rust-infected plants had an unknown function.
This same expression profile was also observed in bean upon rust infection by both races.
After successful colonization of the bean cell, rust race 41 likely secretes effector proteins that can suppress the plant defense pathway induced by PAMPs.
These 90 representative genes mainly identified genes involved in the early responses of the bean under rust infection (i.e., first 96 HAI).
The expression pattern of 90 bean genes upon rust infection shares several features with other legumes infected by biotrophic fungi.
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