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In any case, the validity of the conclusions of tissue engineering will have to be verified in human clinical models using methodologically correct prospective epidemiological designs.
Although these results are promising they need to be verified in human studies.
Although our findings from experimental RA mouse models remain to be verified in human RA pathogenesis, we carefully suggest new insights into the effects of chondrocytes on synovial cell motility into the cartilage pannus interface in RA pathogenesis and highlight the relevance of HIF-2α as a potential novel therapeutic target in RA.
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However, these observations should be verified in humans.
First, the findings in this study need to be verified in humans.
Furthermore, most of the studies have been performed on laboratory rodents and need to be verified in humans.
The linearity of the detector response was verified in human plasma over the concentration range 0.100 200 ng mL−1.
Before cloning the predicted GABAB1k, its existence was verified in human, mouse, and rat.
However, this fact has never been verified in human hydrocephalus.
This effect, however, has not been verified in human glioblastoma cells before.
Increased MMPs activity was verified in human tendons after acute running exercise [ 39] and in exercised rats (jumping in water) [ 14].
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