At the mid-pupal stage (2 3 days post-pupariation), HIG is transported from cell bodies to synaptic terminals.
This chemical is transported from cell to cell, and its flow though the plant is redirected continuously as the plant is developing.
In addition to the above well-characterized trafficking routes, a novel mode of the EGFR signaling pathway, in which EGFR after endocytosis can be transported from the cell surface to different compartments within cells, including the Golgi apparatus, the ER, the mitochondria, as well as the nucleus, has also been reported [ 25, 27, 44].
The cell may also temporarily halt certain processes: for example, newly constructed proteins may no longer be transported from the cell structure where they are made called the endoplasmic reticulum to their final destinations in the cell.
In order to properly maintain these long processes, newly synthesized material must be transported from the cell body to the end of the axon and material must also be recycled back to the cell body.
Apparently, only after sodium has catalyzed the transferal of the phosphate to the enzyme can it be transported from the cell.
Internalized EGFR can also be transported from the cell surface to several intracellular compartments, such as the Golgi apparatus, the endoplasmic reticulum, and the mitochondria, in addition to the nucleus.
As well, the expression of pre-miRs in synaptic fractions implies that the pre-miRs must be transported from the cell body to dendrite shafts and/or to dendritic spines.
As artemisinic acid is likely to be produced faster than it can be transported from the cell, the engineered yeast harboring the plasmid carrying AMO is at a selective disadvantage relative to the same yeast with no plasmid, which results in the low plasmid stabilities observed in non-selective medium.
Efflux enhancement can be described by the 'vacuum cleaner' model [40] which suggests that substrates can be transported from the endothelial cells or brain parenchyma back into the blood.
