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The answer turns out to be that the cells hasten aging in the tissues in which they accumulate.
The problem seems to be that the cells with only one copy of the XOR gene could not replenish their supply of the protein fast enough to sustain membrane production.
It may be that the cells are brought in, from the circulation, or that cells already in the uterine wall divide and differentiate, giving rise to the new lining.
Since cell division is still inhibited at these time points it could be that the cells are not segregating their DNA in the usual manner.
Because we have shown only an effect of CX3CL1 on ROS production by classical monocytes, it may be that the cells recruited are nonclassical monocytes.
It may be that the cells adapt to DRAM-3 loss to survive and/or we have not yet found a condition where endogenous DRAM-3 is critical.
Similar(49)
It may be that the cell phone had something to do with the decline in the phone booth.
The explanation for this could be that the cell sorting methods are not completely efficient.
The result was that the cells, though alive, were unable to mature properly.
The hope is that the cells might be turned into heart cells, brain cells or other types of cells to repair damaged and diseased organs.
The only restrictions are that the cells should not be made into viable embryos or used for human cloning.
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com