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Previous studies on numerosity estimation have reported a tendency to underestimate the actual value [57], [59], although this tendency can be reverted with proper calibration [60].
All somatic mutations outside VH CDR3 in either VH or VL of this antibody, however, could be reverted with no effect on DNA binding.
An interesting characteristic of this oncogenic protein is that its transcriptional repression effects can be reverted with pharmacologic doses of ATRA, resulting in the reactivation of genes essential for definitive myeloid differentiation [ 2, 3, 25].
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After 5 months of Mn-mixture inhalation striatal dopamine content decreased 71%, mice developed evident deficits in motor performance manifested as akinesia, postural instability and action tremor; these alterations were reverted with l-DOPA treatment.
The GPC and wet gluten content increased in untreated plots under 0 kg N ha−1 and 70 kg N ha−1 rate and was reduced following fungicide applications, however, this was reverted with the maximum N rate (140 kg N ha−1).
The effect of MTX was reverted with folinic acid (3 μg/ml).
These insulin component expression levels were reverted with CASP3/7 inhibition in these TNF-α– or IL-6 treated IL-6 treated
The increased iNOS expression in these cells has been reverted with α9β1 integrin blockade, indicating that MMP-9 or uPAR regulates iNOS via α9β1 integrin.
This is based on the observation that dCK deficiency is associated with gemcitabine resistance (Bergman et al, 1999, 2000) which can be reverted by transfection with human dCK (Van der Wilt et al, 2000).
The inactive sulfenamide structure can be reverted to cysteine with reducing agents, thus providing a reversible mechanism of PTPR activation.
The mutant phenotype could be reverted by complementation with a plasmid carrying the coding region of arsR, demonstrating that the observed phenotype was due to arsR.
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com