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First, the most likely reason for this differential pattern is that certain B. p antigens may be recognized differently by the murine and human host immune systems, which contributes to the observed differences.
Finally, motifs may be recognized differently by DNA repair mechanisms (see Harr and Schlötterer 2000; Schlötterer et al., 2006).
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Different forms of hormones are recognized differently by cells, so it makes sense that their effects might also be different.
If antiestrogens are recognized differently by the cell as it has been suggested, then in disease conditions where tamoxifen fails to function effectively, a mechanistically different antiestrogen might yield successful results.
Transcription of AAT repeats and ATT repeats may produce distinct repeats DNA conformations that may differently be recognized by H-NS, and therefore differently affect the IS1E transpositions.
Finally, the fact that the GPI-T signal sequence lacks a consensus sequence but appears to be recognized by various GPI-T orthologues differently raises the possibility that structural variations could participate in dictating sequence specificity.
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DNA polymerases are influenced differently by the factors listed above; therefore, each polymerase generates its unique pattern of errors by which it can be recognized.
These differently methylated histone lysine residues are recognized by different chromatin protein modules that induce distinct chromatin structures.
We show that spontaneously and ligand-induced EGFR activation gives rise to distinct molecular states that are recognized and processed differently by the endocytic machinery.
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