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It is currently well known that cancer is characterised by a tremendous amount of epigenetic aberrations, and hence it could be postulated that these aberrations could have an influence on the immune genes in cancer.
It may be postulated that these models are simply measuring the relative death rates of the Phe/Phe and Phe/Tyr mutants, as compared to EF3030, and not actual survival and growth.
As fluid abilities nearly fully attenuated the association between both the TOFHLA and the NVS with age, it can be postulated that these two constructs have significant overlap with regard to their roles during ageing.
Because of more frequent extracellular localisation of the transcripts within PDR subgroup and the cytokine signaling related subgroup (CSR subgroup) relative to PSR subgroup, it could be postulated that these gene products could directly interact with host proteins.
It could be postulated that these time-dependent increases are caused by vasodilation, increased blood flow and neutrophil migration, however this seems unlikely given that the miRNA increases are seen at 1 hr despite the absence of leukocyte infiltration and miRNAs are decreased at 6 hrs even though neutrophil levels are still elevated.
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It can be postulated that failure of signals through these receptors could affect the antigen-dependent pathway of B cell maturation during which CSR and SHM take place.
It is postulated that these square pores result from crystallographic etching.
It has been postulated that these mutations may represent a fingerprint of aromatic amine which are the most potent class of bladder carcinogens in tobacco smoke.
It has been postulated that these glacial outbursts triggered hyperpycnal flows delivering sediment into the deep-water geologically instantaneously (within days to months)16,30.
It is postulated that these differences are caused both by the different breakwater designs and by variation in longshore sediment supply.
It is postulated that these deviations are attributable to the existence of distributions in flaw size and location, in relation to the bell-shaped tensile stress fields responsible for initiation of the radial cracks at the coating lower surface.
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com