Among persons who have been exposed to influenza virus, the likelihood of contracting disease may be modified by underlying host factors and medical conditions, such as age, smoking status, nutritional status, stress levels, and cardiopulmonary disease.
The results suggest that the haematological response of mice to L1210 leukaemic cells varies according to the route of inoculation, and that these tumour-induced haematological responses of the host can be modified by treatment with VM-26.
E. coli C3081 (NEB) was first transformed by pACYC184-draIIIM so that the DraIII sites in the host genome could be modified by the constitutively expressed DraIII methyltransferase controlled by the Tet promoter.
EHEC and EPEC Tir C-termini appear to be modified by serine/threonine phosphorylation upon entry into the host cell [19], [20].
However, these properties may be modified by the development of discontinuities in the host-rock.
These residues can be modified by protein glycosylation, which is an important posttranslational modification for host-parasite interactions.
However, the same ecological factor may act inconsistently in different geographical areas (Kleinschmidt et al., 2001b; Gemperli et al., 2006; Noor et al., 2008), and the effect of ecological factors is modified by fine-scale vector and host movement (Perkins et al., 2013).
In frequency domain, the transformed coefficients of the host image are modified by the watermark image [1].
Although the molecular mechanisms of CI are currently unknown, several studies suggest that host sperm is modified by Wolbachia during spermatogenesis.
