It seemed that if the contents of ginsenosides were low in primary xylem, the contents would be high in cortex (main root of Pq2 4); and if the contents of ginsenosides were high in primary xylem, the cortex would have a medium (main root of Pq1, Pq5, Pq7, Pq8, Pq10) or low (Pq6, Pq9, Pq11 13) level of ginsenosides.
It is low in primary care but very much higher among women attending STI clinics.
The PPV of the full LFT panel for specific disease affecting the liver/biliary tract is low in primary care.
Its expression is low in primary follicles but increases to maximal levels in pre-antral and small antral follicles (up to 6 7 mm in diameter).
Frequencies of p53 mutations are low in primary MB but Increase significantly in recurrences, and mutant p53 proteins and Myc may collaborate to drive aggressive disease [ 8].
We conclude from these studies that expression of high molecular weight tm isoforms is low in primary breast carcinomas, and that metastatic tumours express relatively high levels of tm 1.
The potential clinical significance of this observation was illustrated by the fact that CD44 palmitoylation levels were low in primary cultures from human invasive ductal carcinomas in comparison with nontumour tissue; and that co-localisation of CD44 with the lipid raft marker Flotillin-1 was also low in invasive specimens.
For children not achieving the PA guidelines in primary school, the mean number of steps on weekdays was low in primary and secondary school and almost no change from primary to secondary school could be observed, while for children achieving the PA guideline at baseline, the mean number of steps in primary school was high but showed a steep decrease from primary to secondary school.
In Scots pine SuSy activity was observed to peak in the zone of maturing tracheids where the secondary wall is formed, and its expression was lowest in primary wall tissues [ 41].
Binding of the two major isoforms of VEGFA (VEGF165 and VEGF121) to VEGFR-2 and VEGFR-1 on endothelial cells was lowest in primary (localized) tumors, whereas the collective binding of the class 3 Semaphorins to endothelial plexins decreased in primary tumors relative to normal tissue and decreased further in metastatic tumors (Fig. 5a).
