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Much remains to be learned regarding the etiology and biological treatments of complex headaches.
Despite great progress in understanding the utilization of CRISPR/Cas9 in a variety of model organisms, much remains to be learned regarding the efficiency and specificity of CRISPR/Cas9-mediated gene editing in human cells, especially in embryos.
However, despite its attractiveness as an efficient, multi-system countermeasure and its potential for simplifying operational activities, much still needs to be learned regarding the human response to rotating environments before artificial gravity can be successfully implemented.
This suggests that there is still much to be learned regarding the biological consequences of T cell entry into tumor microenvironments.
Clearly, much more needs to be learned regarding the complex interactions that are possible between TRBP and its potential binding partners before these effects can be explained.
Much therefore remains to be learned regarding the activating roles of posttranslational modifications in HSF1 regulation, their role in stress mediated transcription and the mechanisms by which such modifications are recognized by regulatory pathways in cells and converted into altered function.
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Many important lessons were learned regarding the creation, engineering and operation of such subsurface systems these lessons will prove valuable as the geothermal community moves towards the goal of realizing the immense potential of this ubiquitous renewable energy resource.
Much has been learned regarding the biochemical characterization of the histone methyltransferase (HMT) activities of the SET domain proteins and their effects on both gene repression and gene activation [13], [14].
This disparity is opposite the situation with respect to carbohydrate-binding activity, in which much more has been learned regarding the galectins.
Although a great deal has been learned regarding the genetic changes that give rise to adaptation in bacteria and yeast, an understanding of how new complex traits arise in multicellular organisms is far less complete.
As more is learned regarding the normal functions of polycystins and how mutations in them disrupt normal cell physiology, the ability to design therapeutic interventions based on gene function and specific pathophysiological mechanisms may progress.
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com