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Thus, regulation of endogenous neuroprotective factors may be key for cell survival and attenuation of disease progression.
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Loss of EGFR signaling, which prevents cells from differentiating, causes these cells to have prolonged noisy Yan expression, and suggests that Yan noise is key for cell state transitions in the eye.
Molecular motor regulated active contractile force is key for cells sensing and responding to their mechanical environment, which leads to characteristic structures and functions of cells.
Conformationally flexible compounds can, however, combine high permeability and solubility, properties that are keys for cell permeability and intestinal absorption.
Therefore, investigating the dynamics of such GRNs may be key for understanding cell differentiation, cell patterning and morphogenesis during developmental processes.
Histone deacetylase 1 3 and HDAC8 have been demonstrated to be key for tumour cell proliferation (Glaser et al, 2003).
The trimer:dimer ratio is key for EBV cell tropism; EBV from B cells tends to infect easily epithelial cells whereas EBV from epithelial cells efficiently infects B lymphocytes (Borza and Hutt-Fletcher 2002).
For example, the cyclin-dependent kinase recognition motif [ST]Px[KR] is present in many mitotic proteins, and its phosphorylation is key for regulating cell cycle progression.
Not surprisingly, control of the quantity and quality of gene expression is key for the cell morphology establishment and cellular signal transduction.
Ion channels have a central role in regulation of the ionic composition of intracellular and extracellular fluid, which are key for hair cell activation in the inner ear.
Histone deacetylase 1 is key for tumour cell proliferation, and found to be upregulated in hormone refractory prostate cancer and breast cancer (Kawai et al, 2003; Halkidou et al, 2004).
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CEO of Professional Science Editing for Scientists @ prosciediting.com