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On the other hand, it can be hypothesized that highly exposed or distressed survivors would be less likely to respond because they do not want to be reminded of the stressful event.
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It has been hypothesized that highly triaxial residual stress fields may be sufficient to promote creep damage in thermally aged components, even in the absence of in-service loads.
It is hypothesized that highly specific antibody-conjugated SPIO would enable MRI of inflamed endothelial cells.
Previously, it was hypothesized that highly acute toxic compounds have little room to increase through prolonged exposure times [2].
Based on these observations, it has been hypothesized that highly pathogenic avian viruses cause a "cytokine storm" which results in detrimental immune responses, although this has recently been questioned in connection with H5N1 virus [53].
It is hypothesized that highly expressed genes may experience strong selective forces toward gene compactness, due to the energetic costs associated with transcription processes (4, 5).
Therefore, it was hypothesized that: H1.
It can be hypothesized that, either the electrostatic interactions of the highly cationic C-ter domain with the negatively charged N-ter domain and extracellular loops of CXCR4 [40], or the steric hindrance promoted by the bulky basic residues in the γ-wt C-ter domain, impair the specific interaction with CXCR4 and therefore reduce the agonist potency of γ-wt.
Thus, it may be hypothesized that.
Hence, it may be hypothesized that.
Therefore, it may be hypothesized that.
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CEO of Professional Science Editing for Scientists @ prosciediting.com