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It can be hypothesized that a Grotthuss-type proton transport physiology-concerning mechanism in channel-like, phospholipid water cartilage's articulating nanospaces will be responsible for the expression of the lubrication mode.
It can be hypothesized that a victim who is small, frail, or even under the influence of a substance is more vulnerable and more likely to be targeted by an offender.
Therefore, it can be hypothesized that a direct effect on TG or at central level may occur as well as an indirect activation via exposed keratinocytes or other cells in the nerve endings proximity.
Since sleep loss and REM sleep loss tend to induce increased pain sensitivity in healthy people [8], it can also be hypothesized that a disturbed sleep structure can cause the observed pre-attack hyperalgesia in migraineurs.
First, it could be hypothesized that a too large release of MELM provided by stimulation of both hind paws triggered a presynaptic inhibition of the peptide release.
It could thus be hypothesized that a vaccine inducing potent CD4+ T cell responses might have a protective effect in HIV/SIV infection, possibly due to cognate help, leading to induction, maintenance and differentiation of CD8+ cytotoxic and/or B cell responses.
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Furthermore, it can be hypothesized that an IL-17 phenotype exists that may define patient subpopulations who particularly benefit from IL-17 targeting therapies.
It is hypothesized that a cytokine storm plays an important role in the pathophysiology of SFTS.
It has been hypothesized that a differential inflammatory response may explain this disparity.
It was hypothesized that a combination of factors would explain variance on two measures of insight.
Presently, it was hypothesized that a single testosterone administration to humans would reduce unconscious fear but not consciously experienced anxiety.
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