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The equation k x (ω, x1) = 0 at fixed ω has a solution which can be found graphically as the intersection point of the function Ω PN (x1) curve and the horizontal line for the frequency ω (see Fig. 2).
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Significant over-representation of genes associated with cell adhesion (Benjamini corrected p-value 6.8×10−4) was found, as well as mitotic cell cycle (Benjamini corrected p-value <0.1); graphically presented in Fig. S2.
To assess whether the observed inverse relationship with PA reflected reverse causation due to smoking, we stratified the analysis by smoking status (Table 2), but no variation was found (P interaction=0.48) as shown graphically in Figure 3 comparing never vs current and former smokers.
The assumption of proportional hazards was evaluated graphically; no deviation from proportionality was found.
The optimized geometry and performance are reported graphically as functions of k̃, φ and N. Good agreement is found between the analytical solution and the numerical results.
The proportional hazards assumptions for stress resilience in relation to CHD outcomes were tested graphically, as well as using a test based on Schoenfeld residuals, and no evidence for violation was found.
As graphically illustrated in Figure 2b, a total of 338 genes were found to be differentially expressed in 'Jinnan Shiqin' and 'Ventura'.
When users investigate promoters of known genes, conserved regions of homologous gene promoters are displayed graphically, as are regulatory features found in conserved promoter regions.
All of the results were presented graphically as well as by spreadsheets.
Results are analysed configurationally (graphically) as well as with RV coefficients, semantically and practically.
The subscores can be presented graphically as a HOOS profile (Fig. 1).
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com